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  • Epoxide Hydrolase, a Fat New Target for Alzheimer’s Therapy?
    Inhibiting the hydrolase also prevented neuroinflammation and amyloid pathology, synapse damage, and cognitive decline in the mice Given that epoxide hydrolase inhibitors are already being tested in the clinic for other conditions, they may have the potential to become a new class of AD drug
  • Hepatic soluble epoxide hydrolase activity regulates cerebral Aβ . . .
    Hepatic soluble epoxide hydrolase activity regulates cerebral Aβ metabolism and the pathogenesis of Alzheimer's disease in mice Neuron 2023 Sep 20;111 (18):2847-2862 e10
  • An epoxide hydrolase inhibitor reduces neuroinflammation in a mouse . . .
    Richard Bazinet University of Toronto Posted: 15 Dec 2020 News: Epoxide Hydrolase, a Fat New Target for Alzheimer’s Therapy? Drugs developed for the treatment of Alzheimer’s’ disease have had very limited success, to say the least This study examining a soluble epoxide hydrolase (sEH) inhibitor is a novel and welcome approach for the field The brain is abundant in arachidonic acid
  • Could Inhibiting a Liver Enzyme Ameliorate Alzheimer’s . . . - ALZFORUM
    But what if targeting a peripheral enzyme could do the job? In the June 26 Neuron online, researchers led by Xin-Hong Zhu at Shenzhen University, China, reported that inhibiting a liver enzyme, soluble epoxide hydrolase (sEH), boosted the amount of protective epoxy fatty acids, particularly 14,15-epoxyeicosatrienoic acid, in the blood of mice
  • Genetic deletion of soluble epoxide hydrolase delays the progression of . . .
    Lee HT, Lee KI, Chen CH, Lee TS Genetic deletion of soluble epoxide hydrolase delays the progression of Alzheimer's disease J Neuroinflammation 2019 Dec 17;16 (1):267 PubMed Recommends Please login to recommend the paper Make a Comment
  • GBA | ALZFORUM
    GBA encodes glucocerebrosidase, a lysosomal hydrolase that digests glycolipids When the enzyme’s activity is impaired, these lipids build up in lysosomes, leading to cellular damage and inflammation As PD, PDD, and DLB are diseases of α-synuclein aggregation, the link between GBA and α-synuclein is under intense study
  • TMEM175 Maintains Lysosome pH by Pushing Out Protons
    Indeed, in TMEM175 knockout cells, the lysosomal hydrolase cathepsin B cleaved less fluorogenic substrate Expression of wild-type TMEM175, but not the D41A mutant, restored cathepsin B’s activity, hinting that too much lysosome acidification harms hydrolase activity
  • NB-4746 | ALZFORUM
    NB-4746 is an oral, brain-penetrant, small molecule inhibitor of the nicotinamide adenine dinucleotide (NAD) hydrolase SARM1 SARM1 activation is required for axon degeneration after nerve injury, and SARM1 inhibitors have the potential to prevent or slow neurodegenerative diseases that involve axon loss





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