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  • CCL3 - Wikipedia
    CCL3 is a cytokine belonging to the CC chemokine family that is involved in the acute inflammatory state in the recruitment and activation of polymorphonuclear leukocytes [6] through binding to the receptors CCR1, CCR4 and CCR5
  • CCL3 Gene - GeneCards
    CCL3 acts through binding to CCR1, CCR4, and CCR5, linking receptor engagement to inflammatory responses and innate immune signaling It is a major HIV-suppressive factor released by CD8+ T cells and inhibits HIV-1, HIV-2, and SIV replication
  • CCL3 - an overview | ScienceDirect Topics
    CCL3 is a protein that directly stimulates the formation of cells that break down bone (osteoclastogenesis) and can also enhance the expression of other proteins involved in bone metabolism
  • CCL3 is produced by aged neutrophils across cancers and promotes tumor . . .
    Through mechanistic perturbations of neutrophil-derived CCL3 in mice, we show that it sustains TAN survival in hypoxic tumor regions via CCR1-dependent signaling
  • The Chemokine CCL3 Regulates Myeloid Differentiation and . . . - Nature
    In murine models of chronic myelogenous leukemia (CML), CCL3 is critical for the maintenance of a leukemia stem cell population, and leukemia progression
  • CCL3 - C-C motif chemokine 3 - Homo sapiens (Human) | UniProtKB
    Monokine with inflammatory and chemokinetic properties Binds to CCR1, CCR4 and CCR5 One of the major HIV-suppressive factors produced by CD8+ T-cells Recombinant MIP-1-alpha induces a dose-dependent inhibition of different strains of HIV-1, HIV-2, and simian immunodeficiency virus (SIV) Showing features for site
  • CCL3 Enhances Antitumor Immune Priming in the Lymph Node
    Our results revealed that CCL3-dependent IFNγ production and CCL3-induced DC maturation drive the priming of effective antitumor immunity in the TDLN Immunogenic tumors are capable of triggering robust antitumor immune responses
  • Targeted delivery of CCL3 reprograms macrophage antigen presentation . . .
    Our study reveals that CCL3 can induce the formation of tertiary lymphoid structures, reprogram macrophages, and enhance antigen presentation mechanisms mediated by the CCL3-CCR5 pathway, thereby improving adaptive immune responses
  • CCL3 is produced by aged neutrophils across cancers and promotes tumor . . .
    Using this framework, we identify CCL3 as a conserved marker of terminally differentiated, pro-tumor neutrophils in growing human and mouse tumors Targeted perturbation studies in mice show that neutrophil-derived CCL3 supports TAN survival in hypoxic tumor niches, thereby promoting tumor growth





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