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  • SMAD Proteins in TGF-β Signalling Pathway in Cancer . . .
    In addition, OSR1 acts directly on SMAD2 3 and phosphorylates 223Thr in the SMAD2-linked region and 179Thr in the SMAD3-linked region, promoting TGF-β pathway signalling and breast cancer metastasis In triple negative breast cancers, growth differentiation factor-10 (GDF10), a member of the TGF-β superfamily, was downregulated in tumour
  • TGF-β induced EMT and stemness characteristics are . . . - Nature
    The effects of retinoic acid and MAPK inhibitors on phosphorylation of smad2 3 induced by transforming growth factor beta1 TGF-beta and stem cell pathways in tumor cell invasion: role of EMT
  • TGF-β signaling: A recap of SMAD-independent and SMAD . . .
    Transforming growth factor-β (TGF-β) is a proinflammatory cytokine known to control a diverse array of pathological and physiological conditions during normal development and tumorigenesis TGF-β-mediated physiological effects are heterogeneous and vary among different types of cells and environment …
  • RNA regulatory mechanisms controlling TGF-β signaling and EMT . . .
    MiRNA targeting of the TGF-β SMAD signal transduction pathway TGF-β Receptors (TGFBR1, TGFBR2), R-SMADS (SMAD2 3, SMAD1 5 8), co-SMADs (SMAD4) and inhibitory SMADs (SMAD7) are directly targeted by each of the listed miRNAs and lncRNAs LncRNAs that exert a positive effect on TGF-β signaling are shown in green; negative regulators in red
  • The role of TGF-β SMAD4 signaling in cancer - PMC
    (A) RAS RAF ERK1 2 axis regulates the TGF-β SMAD4 pathway by (1) phosphorylating SMAD2 and SMAD3 to prevent its translocation into the nucleus; (2) mediating SMAD4 degradation; and (3) promoting AP-1 complex formation at the TGF-β1 promoter, thus boost the TGF-β1 transcription and secretion As the substrate of JNK, c-Jun can directly bind
  • TGF-β and the Smad Signaling Pathway Support Transcriptomic . . .
    Epithelial-mesenchymal transition (EMT) contributes to normal tissue patterning and carcinoma invasiveness We show that transforming growth factor (TGF)-β activin members, but not bone morphogenetic protein (BMP) members, can induce EMT in normal human and mouse epithelial cells EMT correlates with the ability of these ligands to induce growth arrest Ectopic expression of all type I
  • SMAD Proteins in TGF-β Signalling Pathway in Cancer . . . - MDPI
    Suppressor of mother against decapentaplegic (SMAD) family proteins are central to one of the most versatile cytokine signalling pathways in metazoan biology, the transforming growth factor-β (TGF-β) pathway The TGF-β pathway is widely known for its dual role in cancer progression as both an inhibitor of tumour cell growth and an inducer of tumour metastasis This is mainly mediated


















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