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  • Activation of chaperone-mediated autophagy as a potential anticancer . . .
    In a recent study, we provide a novel mechanism by which excessive activation of CMA can be exploited as a method to eliminate cancer cells by inducing metabolic catastrophe and delineate a novel strategy to promote the degradation of HK2 (hexokinase 2) in cancer cells
  • Activation of chaperone-mediated autophagy as a potential anticancer . . .
    In a recent study, we provide a novel mechanism by which excessive activation of CMA can be exploited as a method to eliminate cancer cells by inducing metabolic catastrophe and delineate a novel strategy to promote the degradation of HK2 (hexokinase 2) in cancer cells
  • Rewiring chaperone-mediated autophagy in cancer by a prion-like . . .
    In this study, bioinformatics analysis revealed the co-occurrence of upregulated CMA and PD-L1 accumulation in metastatic melanoma with adaptive immune resistance (AIR) to anti-PD1 treatment, suggesting the potential therapeutic effects of rewiring CMA for PD-L1 degradation
  • Targeting Chaperone-Mediated Autophagy in Cancer - Science
    Selective inhibition of CMA slows tumor growth, boosts tumor cell death, and induces the regression of existing tumors This work provides a rationale for further exploring manipulation of the CMA pathway as a strategy for treating cancer
  • Activation of chaperone-mediated autophagy as a potential anticancer . . .
    In a recent study, we provide a novel mechanism by which excessive acti-vation of CMA can be exploited as a method to eliminate cancer cells by inducing metabolic catastrophe and delineate a novel strategy to promote the degradation of HK2 (hexokinase 2) in cancer cells
  • Frontiers | The Role of Autophagy in Tumor Immunology—Complex . . .
    Effective Antitumor Immune Responses Effective antitumor responses are dependent on potent antigen presentation and leukocyte infiltrates enriched with effector CD8 + T cells () Autophagy activation in DCs may improve antigen presentation and stimulate cytotoxic responses mediated by CD8 + T lymphocytes (47, 49) For example, nano-activators conjugated to antigens were used to stimulate DCs





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